Welkom bij het Lymeforum

Abstract
A 19‐year‐old man developed chronic weakness of the lower limbs as the predominant manifestation of Borrelia burgdorferi infection of the nervous system. Spirochetes were demonstrated in the cerebrospinal fluid. The condition resolved following intravenous penicillin treatment.

Henriëtte schreef:Meningoradiculitis na insectenbeet (syndroom van Bannwarth);

https://www.ntvg.nl/system/files/public ... 60001a.pdf

Henriëtte

Ook zijn de patienten op veel infectieziektes getest en alles negatief, ik kan nergens vinden of er op borrelia is getest.

In een patiëntenonderzoek met 176 neuroborreliose patiënten was bij 86% radiculaire pijn de eerste klacht.
Met betrekking tot cognitieve en neuropsychiatrische (vervolg) ziekten als deel van borreliose is in Nederland nagenoeg geen onderzoek gedaan. Daarom zullen deze patiënten vaak alleen symptomatisch behandeld worden daar niet aan Lyme gedacht wordt. In dit stadium kunnen de volgende aandoeningen zich voor doen:

Meningitis
Meningoradiculitis Bannwarth syndroom
Perifere faciale parese
Lyme carditis
Lyme arthritis
Orchitis(ontsteking van de teelbal)
Uveitis(ontsteking van de iris)
Panoftalmitis(ontsteking van het oor)
Hepatitis
Myositis
Plexusneuritis
Mononeuritis
Encefalitis
Cerebrale vasculitis
Tinteling en doof gevoel in de ledematen zoals Carpal tunnel syndroom
Myelitis

Abstract

Myositis is a rare manifestation of Lyme disease of unknown pathogenesis. This study describes the course of disease in eight patients with Lyme disease, aged 37–70 years, all of whom were suffering from histologically proven myositis.
The Clnical, electrophysiological, and myopathological findings are reported.
One patient showed signs and symptoms of myositis of all limbs. In six patients myositis was localized in the vicinity of skin lesions, arthritis or neuropathy caused by Borrelia burgdorferi.
In another patient suffering from pronounced muscle weakness of the legs and cardiac arrest, inflammation of the myocardium, the conducting system and skeletal muscles was revealed at autopsy.
Muscle biopsy revealed lymphoplasmocellular infiltrates combined with few fibre degenerations in three patients.
The lymphoplasmocellular infiltrates were found predominantly in the vicinity of small vessels. Several spirochetes were stained in six of seven muscle biopsy samples by means of the immunogold-silver technique.
Culturing of B. Burgdorferi from the muscle biopsy samples was, however, unsuccessful.
Antibiotic treatment succeeded in curing the myositis in four of six patients.
In one patients signs and symptoms improved.
One patient died from cardiac arrest caused by myocarditis and Guillain-Barré syndrome.
The outcome is unknown in one patient. Clinical and myopathological findings indicate that Lyme myositis can be caused either by local spreading o fB. burgdorferi or an unknown antigen or toxin from adjacent tissues or haematogenously.